How do most traditional antineoplastic drugs achieve selective toxicity against tumor cells and why is that mechanism of selective toxicity problematic from the perspective of adverse effects?
a. Target cells with tumor cell markers, but adverse effects can occur because these markers are occasionally present on healthy cells as well.
b. Traditional antineoplastic drugs are not selectively toxic at all and can easily cause patient deaths, this is why treatment is moving towards the use of more targeted therapies and precision medicine
c. I have no idea and would rather be eating tacos that working on this assessment......
d. Target cells that are dividing rapidly, but adverse effects can occur because even in adults there are populations of cells that still divide rapidly (bone marrow, mucous membranes, etc.)